Dysfunctional mechanotransduction through the YAP/TAZ/Hippo pathway as a feature of chronic disease

Mathias Cobbaut, Simge Karagil, Lucrezia Bruno, Maria Del Carmen Diaz de la Loza, Francesca E. Mackenzie, Michael Stolinski, Ahmed Elbediwy

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    Abstract

    In order to ascertain their external environment, cells and tissues have the capability to sense and process a variety of stresses, including stretching and compression forces. These mechanical forces, as experienced by cells and tissues, are then converted into biochemical signals within the cell, leading to a number of cellular mechanisms being activated, including proliferation, differentiation and migration. If the conversion of mechanical cues into biochemical signals is perturbed in any way, then this can be potentially implicated in chronic disease development and processes such as neurological disorders, cancer and obesity. This review will focus on how the interplay between mechanotransduction, cellular structure, metabolism and signalling cascades led by the Hippo-YAP/TAZ axis can lead to a number of chronic diseases and suggest how we can target various pathways in order to design therapeutic targets for these debilitating diseases and conditions.
    Original languageEnglish
    Article number151
    JournalCells
    Volume9
    Issue number1
    Early online date8 Jan 2020
    DOIs
    Publication statusPublished - 8 Jan 2020

    Bibliographical note

    Note: This work was supported by the Francis Crick Institute.

    Keywords

    • Biological sciences

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